Holy crap, that’s only 6x7= 42mg a week. I pin 17mg daily for 120mg a week and my SHGB is lower than yours. Crazy how individual TRT is! Good luck! :)))
Any update? I did got similar sensation today as well.
Dr. Saya said what I was describing pointed to an immune system response, cotton seed oil is known to have the most cases of allergic reactions, that’s why all the reputable clinics are using sesame seed and grapeseed carrier oils.
I’m going to be using some leftover ethanate from 1.5 years ago and see if it stops what appears to be an allergic reaction of some kind.
Pharmaceutical company Perrigo uses cotton seed in theirs. I would like grape seed too. I wonder if there is a 200mg/1 ml that uses grape seed. I need to use 200mg/1 ml only cause that’s what insurance approved.
Well, as far as I know males produce testosterone 4…7 mg /day. So not that incredible that such “low” dose is working.
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Son of a bitch, I used ethanate today and have noticed that burning, stinging sensation is getting weaker and weaker as if it’s clearing out of my system. I even increased the dosage.
That year long cough and the acid reflux diagnosis, all gone as well!
I can’t believe I went a entire year of hacking and coughing over some stupid cottonseed oil!
So it’s either pay Defy/Empower for cypionate (sesame seed) or get ethanate from Kaiser for $1 copay.
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Unreal. Wow.
I thought empower also has grape seed
Empower has both sesame seed and grape seed carrier oils in their cypionate. That burning, tingling sensation was causing me to cough.
This cough started about the time I began injecting EOD back in November 2017.
REALLY happy to hear you found the problem, systemlord. Awesome!!! 
Hey man. Does defy recommend subq?
I found a study on thyroid hormones and handling bilirubin on rats where hypothyroidism and hyperthyroidism was induced. Hypothyroidism caused a cholestatic condition with a 50% decrease in bile flow and in bilirubin Tm, and with an increased proportion of conjugated bilirubin in liver and plasma.
Thyroid hormones and the hepatic handling of bilirubin. II. Effects of hypothyroidism and hyperthyroidism on the apparent maximal biliary secretion of bilirubin in the Wistar rat.
Thyroid hormones and the hepatic handling of bilirubin. II. Effects of hypothyroidism and hyperthyroidism on the apparent maximal biliary secretion of bilirubin in the Wistar rat.
Abstract
This study was undertaken in the Wistar R/A Pfd rat to investigate the effects of hypothyroidism and of hyperthyroidism on the maximal biliary excretion ™ of bilirubin and on the concentration and composition of bilirubin in liver and plasma at the end of a bilirubin load. Hypothyroidism caused a cholestatic condition with a 50% decrease in bile flow and in bilirubin Tm, and with an increased proportion of conjugated bilirubin in liver and plasma. This was associated with an increased ratio of bilirubin diconjugates to monoconjugates in bile, liver, and plasma, which can be ascribed to the increased hepatic conjugation activity towards bilirubin and/or to the prolonged retention of bile pigments in the hepatocytes with increased conversion of monoconjugates to diconjugates. Cholestasis induced by hypothyroidism was further characterized by a decreased biliary output of unconjugated bilirubin. The latter phenomenon might represent an indirect effect related to a decreased output of bilirubin monoconjugates with impaired hydrolysis to unconjugated bilirubin; it might also reflect the cholestatic condition with decreased excretion of the unesterified bile pigment as such. Hyperthyroidism resulted in a 1.3-1.4-fold increase in bile flow. The maximal bilirubin concentration in bile decreased 1.3-1.4-fold, so that the apparent maximal bilirubin excretion rate remained unchanged at 115 nmol.min-1.100 g-1, as observed in untreated rats. Hyperthyroidism lowered the bilirubin UDP-glucuronosyltransferase activity, produced a decreased ratio of bilirubin di- to monoconjugates in bile and plasma, and a decreased ratio of conjugated to total bile pigment concentration in liver and in plasma. Similar findings are present in the heterozygous Gunn rat strain and in patients with hepatic bilirubin UDP-glucuronosyltransferase deficiency. We therefore propose the hyperthyroid rat as an experimental animal model of Gilbert’s syndrome
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Hey systemlord. Can you please tell me more about your insulin resistance? Symptoms, diagnosis? I suspect this may be ma issue as well. Did you try cinnamon or berberine? Please advise.
I don’t know that I have any symptoms from the insulin resistance as my A1C is 6.0, berberine does nothing.
So you diagnosed it only by A1C? 6.0 is in normal range. I believe I read somewhere that you complained about that issue.
An A1C of 6.0 isn’t optimal, a healthy target is under 5.0. I’m actually closer to pre-diabetes.
ok you right I just read that as well. So no crashing symptoms (low sugar) etc?
Never low blood sugar, no crashing. I believe doctors are treating a numbers, they see the labs, “oh you have diabetes”, I never complain of any symptoms other than low testosterone.
Doctors get paid to diagnose you with a disease, this is why they do not offer treatment when you are what they consider borderline. Once diagnose you with a disease, you are reliant of the pharmaceutical companies for life.
Ok my apology, I swear reading something on this topic by you before, thus the question.