I’m assuming this is a troll, however I’m going to reply anyway, if you seriously think that an optimal cycle involves just orals, then you have decided to run a cycle without doing any research at all. Trenbolone isn’t an oral steroid either, unless you’re ordering cattle implants and taking them by mouth, in which case… ew.
Most highly effective “Prohormones” are just designer steroids, a prohormone is a precursor to an active substance, and many prohormones share characteristics with anabolic steroids. Androstenedione is a prohormone that converts to testosterone via the 17B(however I put in the B it’s a latiny word I think) hydroxysteroid dehydrogenase enzyme and some will also convert to estrone via interaction with aromatase. This is a prohormone. Epistane (methylepitiostanol) is an active anabolic steroid/ derivative of dihydrotestosterone, it doesn’t convert to anything when taken, due to it being a DHT derivative it isn’t converted to anything via 5a reductase, it also doesn’t aromatise (as a matter of fact the non C17AA version of this drug is apparently a medication to treat ER positive breast cancer as it is highly anti-estrogenic, whether the C17AA version carries the same traits is unknown as methylepitiostanol is a designer steroid with little research behind it, one has to go by anecdotal experiences. Most of these “prohormones” such as epistane (that are actually designer steroids) are highly toxic to the liver, they are C17AA (an alteration in the 17th carbon position of the steroid molecule that makes the substance unable to be broken down by the first liver pass following oral ingestion) this alteration, for whatever reason makes all compounds with this alteration toxic to the liver, liver enzymes will be elevated with use and with prolonged use life threatening liver dysfunction can develop, serious liver dysfunction from short term use is possible but unlikely, and if it does happen it’ll usually be in the form of acute cholestasis. Due to the fact that adequate liver function is required for optimal processing of cholesterol (estrogen is also required), C17AA anabolic steroids tend to exhibit a far more detrimental effect on the lipid profile than anabolic steroids without this structural modification, dihydrotestosterone derivitaves also tend to be harsher on the lipids, epistane is a DHT, it’s potentially anti estrogenic and its C17AA, so watch out for lipid strain. An example of a combination of a PH/ designer steroid which is on the market is methyl – 1 – androstenediol (or androstenedione I forgot), it’s a precursor to methyl-1-testosterone, however it appears to have anabolic activity in it’s unaltered form, so while some will convert to M-1-T (an extremely powerful and toxic hormone), M-1-AD is anabolic itself, although I’d never recommend anyone use this stuff. Although M-1-T is commonly thought of as mystical a fun fact is if looked at structurally it’s just 5a reduced DBOL, and DBOL is C17AA EQ, (methyldihydroboldenone) is M1T.
If I was you I wouldn’t fuck around with DS or PH’s, I’m not recommending a first cycle because I don’t feel comfortable doing so, however if this post isn’t a troll I’m sure someone will chime in and help out.