I pack fill syringes JUUUUST before I inject, because otherwise it’s hard to determine exact doses of primoteston (as they’re pre-filled, unmarked syringes)
Adequate androgenic stimulation is also required for optimal sense of wellbeing, DBOL in theory actually might work (however still terrible idea for solo cycle), DBOL itself isn’t very androgenic however it’s 5a reduced metabolite (methyl-1-testosterone) is quite androgenic… and toxic.
I agree the test base is brosciency to some extent, however it’s still reccomended as most AAS people use are unsuitable to be used without testosterone (to provide adequate estrogen and androgen replacment). Nandrolone (not androgenic or estrogenic enough), boldenone (not androgenic enough), drostanolone (no aromatization), methenolone (no aromatization) etc just don’t cut it. I will say this, interestingly enough, fluoxymesterone (HALOTESTIN: an extrordilarily androgenic, yet non aromatizing AAS has and is still FDA approved for treatment of male hypogonadism)
Also different AAS have complex effects on various neurotransmitters (many compounds are relatively untested on humans, thus we don’t know the effects) testosterone is known to increase dopamine and serotonin, 19-nors decrease seratonin and deplete dopamine, thus 7a-methyl-19 nortestosterone is likely not the greatest candidate for TRT. What effect does androgenic and mysteriously supposedly estrogenic compound like oxymetholone have with regard to affects on neurotransmitters?