[quote]eaboadar wrote:
Interesting article. Yes, the progression of “T” levels during an after treatment is strange. The HCG clearly induced the testes to start producing Testosterone again. During this time the “top” of the HPTA should have remained suppressed. When HCG was ceased the testes were now stimulated anymore so Testosterone levels dropped initially but there was something that made the hypothalamus and pituitary to start working again? Any thoughts? Maybe the pituitary and/or hypothalamus got used to “seeing” LH (HCG, actually) and when it was taken away the contrast made it/them react?
Anyway, any reason not to like HCG aside from people taking too much and making their LH receptors insensitive to LH?
Don’t you think one would do well not to allow the testes to become atrophied? Isn’t it counterproductive to recovery?
Thanks and sorry about all the questions. This discussion is very interesting.[/quote]
i think what happened, was that even though the HCG kicked in the testes, the rest of the HPTA still wasn’t ready, and that’s why there was the lag time. i think using a SERM, or even just waiting it out, would have been better in that instance…
my main issues with HCG are the concerns over desensitization, and the user feel they are recovered when they’re not, and not following PCT through…
i’m basically going with a trade-off with my theory… preventing testicular atrophy might help recovery, but it also might delay it, with HCG overuse.
if one felt that they needed to use it, then i would use it close to PCT, and maybe even into the first week or so (along with Nolvaex and Aromasin)…that way one could obtain the benefit of the increased testicular function to go with the HPTA recovery…