I’m going to write a detailed reply in a few hours soley because I find your username amusing, but first I want to take a nap, this post will be edited in the near future.
You need to figure out which ester of testosterone you want to use, test prop will be fine for an 8 weeker but test enth and test cyp need at least ten weeks, preferably twelve. The reasoning behind this is because test prop has a much shorter half life than test E or test C, therefore it reaches peak levels in the bloodstream much quicker. Test E or Test C, like @getcutgetbutt said, should be run at around 500mgs weekly for a first cycle, given at doses of 250mg injected e3.5 days, test prop needs to be injected at least Every other day, but every day would give more stable levels (and test prop is a far more painful injection).
Don’t taper up and down testosterone, just run a determined amount for the whole cycle, in medicine people are taught to taper up medicine to get the desired effect and then taper down in order to wean the patient off medication. When it comes to anabolic steroids, this is an inefficient practice, starting with a dose of 100-200mg is a mere replacement dose, therefore there is literally no point in starting at such a low dose. As for tapering down, all it does is prolong the amount of time you remain shut down, some doctors have the notion that tapering patients off AAS is still an efficient practice, as a matter of fact you can still find this advice in endocrine guidelines on how to taper people off who are abusing AAS, this is a stupid and outdated practice that stems from the fact people are tapered off most medications such as corticosteroids and opiates etc.
Going off AAS cold turkey (the way it’s meant to be done) will put the user at a very low testosterone level for a prolonged period of time because steroids sent a negative feedback loop to the anterior pituitary gland, sending them a message that the body has enough testosterone. The anterior pituitary then shuts off production of LH and FSH (leutinizing hormone and follicle stimulating hormone), FSH and LH are the two hormones that stimulate the leydig cells of the testis to produce testosterone, without LH and FSH the body stops producing testosterone, resulting in testicular atrophy and arrested spermatogenesis, this is where HCG and PCT comes into place. HCG is a LH minick, it tricks the body into thinking it is still producing LH, therefore the body keeps producing intratesticular testosterone, this keeps the testis at their original size and preserves fertility given the individual is fertile to begin with (like me). HCG, being a MIMICK of LH is still suppressive though, once the user goes off LH will crash back down to near zero, however the theory is that if the body is kept producing testosterone, recovery should be quicker. PCT (nolvadex and clomiphene) stimulate the production of the bodies natural FSH and LH, therefore the body starts producing testosterone again at a far quicker pace than what would happen without PCT. With this knowledge you will realize the drug needs to have exited the body before recovery can properly begin, therefore tapering down is a practice that needs to be stopped. The two most commonly used SERMS are nolvadex and clomiphene, for a first PCT i’d use nolvadex as clomiphene can have some really nasty mental side effects, and rare side effects of clomiphene can even include ocular nerve damage resulting damaged or distorted vision
With supraphysiological levels of testosterone (supraphysiological is generally determined as three times the upper limit of normal) (say 2500-3000)ng/dl, and pharmacological is generally considered a dose that produces a result that produces results that wouldn’t happen normally. I’m not sure if this definition of supraphysiologic and pharmacologic refers to AAS, however if you look at some medical literature you can see 300mgs of nandrolone decanoate weekly or 300mg of Test E is considered “a pharmacologic dose”.
Furthermore, with supraphysiologic levels of T, supraphysiolgic levels of estrogen accompany, the amount of aromatization is different for everyone, SHBG levels, genetics, bodyfat each play a role. 5-7 percent of testosterone is aromatized into dihydrotestosterone, this is the androgen responsible for androgenic side effects such as body hair, acne and hair loss. Estrogen related side effects include water retention, high blood pressure, fatigue, moodiness, gyno etc. Estrogen can be controlled by a SERM such as nolvadex, nolva binds to the receptors that are sensitive to estrogen such as breast tissue to stop a person from getting gyno, but doesn’t actually reduce levels of estrogen in the body. Secondly there are aromatase inhibitors to reduce levels of estrogen in the body, there are steroidal and non-steroidal aromatase inhibitors and suicidal and non suicidal aromatase inhibitors. Steroidal AI’s have a steroid backbone (like formestane) whereas non steroidal ones don’t (think anastrazole). Suicidal AI’s permanently bind to the aromatase enzyme, killing it, therefore once usage is stopped there is no chance of an estrogen rebound, non suicidal AI’s temporarily deactivate the enzyme, allowing for a sudden rebound/spike of estrogen when the drug is stopped (this isn’t guaranteed, just possible) AI doses differ for everyone, take it slow, because tanking your estrogen can be more harmful than having high estrogen (Joint pain… JOINT PAIN DAMMIT).
What are your stats, years lifting, age, are you willing to accept the possible long term risks of AAS use such as heart damage in the form of hypertrophic cardiomyopathy, even if the risk isn’t very high it is still a risk you need to know about and understand, you could be looking at never recovering and being on TRT for the rest of your life, I would talk about cholesterol and liver issues but it doesn’t seem as if you are looking at running any orals. Be careful and do as much research as possible, hormones aren’t something you want to play around with, they are serious drugs and like all drugs when abused can have serious consequences. If you have any questions feel free to ask. I personally think you need to do way more research before you consider taking up AAS usage. What is the purpose of wanting to cycle, are you trying to become a competitive athlete or is it for purely aesthetical reasons (serious question, I won’t judge either way, I’m just trying to figure out the reason)