WideGuy....why can't you take everything I say as fact?! It would be so much easier. I'm kidding of course.
Well, I wouldn't speak of it restoring testicular size as such a matter of fact. I've had guys e-mail or tell me in person that they swear their testicles atrophied the day after they initiated a cycle with testosterone. Similarly I've had them tell me their testicles get larger during a cycle. As you know, the testicles aren't going to atrophy that quickly. I think the main part of confusion stems from guys evaluating the size of their scrotum, rather than actually palpating the testicles and getting a physical measurement. Just to give you an example of what I'm talking about, after 6 months of taking testosterone enanthate (normal men) at 300 mg/week, testicular size remained within 0.5 centimeters of pretreatment measurements. That is why I don't put much stock in to such reports as I've never met a guy who actually measured his testicles, nor was able to eye such small changes. Rather, they simply look down here and there.
I'm not sure what you mean about it not stopping gains. There's no data which supports the statement that any aromatase inhibitors decrease gains. I've heard of guys claiming that they use one drug over another because one doesn't affect IGF-1 levels, yet unbeknownst to them, there are just as many studies showing a decrease as there are showing no effect. Psychology plays a large role.
As for the effect upon testosterone levels. I don't doubt you there, but that's not what needs to be measured. Rather, LH needs to be measured. Depending upon which analytical method is used for testosterone, that steroid you're using or a metabolite thereof could be mistaken for testosterone. This is why it's so important to have LH assayed, yet, coincidentally or not, the highly publicized pilot studies for these steroidal aromatase inhibitor supplements lack an evaluation of LH. An evaluation of LH will truly allow one to know if "recovery" is taking place or if the steroid being administered is cross-reacting with the standard immunoglobulin. Bill Roberts has addressed this thoroughly. http://www.t-nation.com/readTopic.do?id=557245
Last, you said that these people were using other supplements when they had their blood drawn which really makes it difficult, if not impossible to differentiate which was responsible for what.
Keep in mind, I'm not saying you're imagining things or that you're wrong, I'm just pointing out why some people could be making such claims. In the end, if it worked for you, by all means, keep doing it.