More doesn’t usually equate to better, that is def true. Higher doses of AAS = more side effects (short + long term), some may not be able to tolerate higher doses. AR saturation theoretically does occur at SOME dose (however given lack of clinical data the dose in which AR saturation occurs is unknown, esp since AAS tends to create new AR in muscle tissue, thus the dose of saturation would have to be high enough to superceed the rate in which new AR could be synthesized.
However I will say this (250mg/wk + 50mg var/day equates to 600mg/wk), that’s not exactly a low dose to me (although many will say “well 600mg is on the low end of cycling”, well yea 600mg isn’t a gram or two however it isn’t “low” either. 250mg/wk +25mg/var/day I would consider low
What’s the use for clenbuterol (not a competitive bodybuilder or athlete trying to make a weight cut), Clen + AAS equates to a higher liklihood of cardiac alterations occuring. 250mg/wk gave me bacne, TRT gives me bacne, some people are just sensitive to (some) androgenic side effects. Given my families genetic profile I’m not particularly likely to go bald, however I have a TON of body hair, always have and I’m prone to bacne. Anabolic steroids cause hypertrophy of the sebacious glands, causing an increase in the production of sebum. Increased sebum production = more acne, no way around that if you’re prone to acne other than accutane (not reccomended # joints # hepatotoxicity # triglycerides), or vigorous self hygiene protocals.
Clomid’s isomer, zuclomiphene is inherently estrogenic and antigonadotrophic, it is the isomer likely responsible for the nasty side effect profile of clomiphene, if you’ve used it and can handle it… great, otherwise don’t use clomid (just a suggestion).
Free + total T
Cholesterol (HDL/LDL, if paranoid like me you can get HDL and LDL subtypes tested as well as VLDL)
Exactly the same