For the fish oil question, there is a good couple of articles by Berardi on this site about them.
However, unless you have a blood thining condition, there really is not an actual limit on the amount you can take beyond the adverse effects it could have on your caloric intake.
Technically, ‘‘primitive’’ nutrition had a significantly higher content in EFAs than what presently the norm in developped countries (less fish and less if any free-ranging animals who have better lipid profils in their meat).
Most people seem to go between 6-12 g (with the same concentration of EFAs you are currently taking or a bit more depending on the company, mine is 180mg EPA, 120mg DHA with recommendations of 3 to 6 per day). But again, EFAs could constitute a very significant portion of your fat intake without much adverse effects (many anti-depressant and increased cognitive fuctions are being investigated right now, so that’s all good also).
One of the recommendatins I saw for fat intake was a maximum of 30% in the ‘‘recommended’’ diet with 10% max for saturated, 10% max from mono-unsat and 10% max from poly-unsat fats. But then again, these are recommendations based on absence of evidence so they couldn’t really recommend to eat a lot of anything, they only thing at that point that the scientific community knew was that saturated fats have negative effects on health. So 10% of 2500 Cals for poly unsats is some 28 g of EFA…which in my case would imply some 93 capsules a day (28g of EFA and 65 of other fats in the capsules)
Like I mentionned above, probably the only real option would be to increase your niacin intake with ordinary, not extended release formulas. Beyond that, you my friend, are stuck with relatively bad genes in that department and medication is the only way to go.
Also, maybe a switch to Crestor as a statin might be considered, since its a more power LDL lowering and more HDL increasing then the lovastatin you are presently taking, which would imply that you would have to go back to a 2-pill regimen instead of the 2-in-1. It might be more expensive also, considering its ‘‘newness’’. The effects would probably be moderate with a limited increase in ‘‘possible’’ liver toxicity, just something to look into.
Something you might want to look into since if you can’t increase your HDL significantly, you might try to reduce your LDL as low as possible to modify your HDL/LDL ratio in a favorable manner (that’s why the Crestor might be interesting since it seems to bit more potent on a mg per mg basis than previous statins.
Hope any of this can help.
Since you are at risk for CHD, maybe looking into all those little thing such as aspirin, green tea, psyllium fibers, cinnamon, increased fish consumption (other benefits beyond EFA in its amino acid profile, mainly increase arginine as a NO precursor for improve endothelial function as opposed to high-methionine meat)…getting all the benefits you can get.