A preface to my questions
once Re-feeds and cheat meals where done on some vague scientific data about re-stimulating metabolism, re-stimulating thyroid functions, phsychological(sp??) stuff etc. their content was anything from whole cows to poor innocent bystanders. no specifics.
now it’s done on the (disputed) notion that through stimulating leptin production, we can jump start the fat loss which stopped due to reduction in serum leptin levels
Although john berardi is opposed to refeeds, from his article on leptin I gathered that leptin production is correlated directly to fat gain due to carbohydrate metabolism to triglycerides and storage of the last ones. since none of that happens (hopefully) when we diet we need to stimulate it ourselves.
So how much fat do I need to gain before I can go back to dropping fat? I dont think anybody can answer that right now.
I didnt see anything in JB’s article that says refeeds are only for lean people or that serum leptin levels are related to the number or size of adipose cells. so why do all the recommendations for refeeds say that they should start at a low BF? if someone hadnt experienced a fat gain due to carbohydrate metabolism to tryglicerides and storage of those tris wouldnt he be subject to the same drop at serum leptin levels, no matter how fat he is?
Question B - Refeeds and Hot-roxx:
On the “we made ephedra obselete” it is said and I quote “Additionally, both increased cAMP and high T3 levels decrease lipoprotein lipase activity, which is an enzyme responsible for lipogenesis. So in addition to stimulating lipolysis, cAMP and T3, as a team, put the kibosh on lipogenesis as well.” so we shouldnt take hot-roxx with our refeeds otherwise we will not gain fat enough for leptin to be produced?
Is the metabolism of glucose to tryglicerides BY ITSELF a leptin stimulator or do they have to be actualy stored? if the answer is yes then it answers question B right?
why do I ask that?
Bill roberts said in the hot-roxx forum, “refeeds and hot-roxx thread” this: “Decreasing tendency towards lipogenesis in the refeed would be a good thing” so he is implying that this might be true. otherwise he contradicts what JB is saying.
thanks for anyone who could clarify