Growth continues until estrogen levels increase, causing the growth plates to close. Then it is all over. This is why women are shorter than men. I just read something about this, again, and there were two cases of abnormally tall stature, one where there was a mutation in aromatase that made it ineffective and another where the was an estrogen receptor defect. Very much locking in the E connection.
There has been medical research discussion of AIs been used to allow for greater stature in males. Severely depressing E in females might have some unexpected serious consequences.
I think that this would work. But when the growth plates close, nothing can be done to allow bone growth after that. By the time that many would be concerned that they were not going to have normal height, it would be too late and what kids would have any knowledge of any of this?
This “suicide aromatase inhibitor” is crap. If not on gear, .5mg of anastrozole per week might be all that is required. Do the math.
Many of these ‘alternative’ AIs are not absorbed very well at all and for some, effectively not at all. The research that “support” claims of effectiveness are often in vitro or IV delivery to study the mode of action. Unfortunately, these sweep the issue of absorption under the rug. The research is often of academic interest and was not intended to determine effectiveness or dosing. But often an industry springs up that takes such data as a marketing tool for things that really do not work well or at all. Even LEF.org is guilty of that!
I had a TRT guy that I gave advice to, go off and get that stuff instead of the anastrozole I recommended earlier… the claims for the “suicide aromatase inhibitor” were too sexy for him to ignore. He later PM’d me stating that the product was expensive to use and that his labs showed little or no E2 changes. We got that fixed. A lesson hard learned by one guy - take note. Many ‘alternatives’ do not work well and cost more than the real thing. Availability is an other issue.