"as if every top tier bodybuilder doesn't use the hell out of orals"... you are aware that they also "use the hell out of" support supplements and "use the hell out of" doctor visits right? Even still, Big A claims that they are basically... incredibly unhealthy despite the measures they take to preserve themselves..
you are aware that PMCChris (from PM) just had an hepatic adenoma the "size of a grapefruit" (or did he say volleyball? I don't remember) removed from his liver correct? They also took a sizeable part of his liver with it..
and my "don't use orals" suggestion was advice that I received from a few IFBB pros.. so.. they also have all said to "control your estrogen".. you should read Kaladryn's posts sometime... he is very smart. He and Shelby are co-authoring a book about health for bodybuilding
well anyways, here is some food for thought. This is why you want to control your estrogen... this may be hard to wrap your head around.. but gyno isn't the only bad thing that elevated estrogen levels can cause..
The liver is morphologically and functionally modulated by sex hormones. Long-term use of oral contraceptives (OCs) and anabolic androgenic steroids (AASs) can induce both benign (hemangioma, adenoma, and focal nodular hyperplasia [FNH]) and malignant (hepatocellular carcinoma [HCC]) hepatocellular tumors. Hepatic adenomas (HAs) are rare, benign neoplasms usually occurring in young women, the development and the complications of which have been related to the strength of OCs and the duration of their use. HA incidence has fallen since the introduction of pills containing smaller amounts of estrogens. FNH is a benign lesion, most commonly seen in young women, which is thought to represent a local hyperplastic response of hepatocytes to a vascular abnormality. Because of the female predominance and the young age at onset, a role of female hormones has been suggested. Furthermore, a large proportion of women with FNH (50-75%) are OC users. Liver hemangiomas (LHs) are the most common benign liver tumors and are seen more commonly in young adult females. The female predilection and clinical observations of LH growth under conditions of estrogenic exposure suggest a possible role for estrogen in the pathogenesis of LHs. HCC has become one of the most widespread tumors in the world in recent years, representing the sixth leading cancer and the third most common cause of death from cancer. Apart from liver cirrhosis, numerous other factors responsible for its onset have been proposed: hepatitis infections from virus B (HBV) and C (HCV), alcohol, smoking, and aflatoxin. However, regardless of etiology, chronic liver diseases progress at unequal rates in the two sexes, with the major sequelae, such as cirrhosis and HCC, being more frequent in men than in women. These epidemiological data have prompted researchers to investigate the relationship between sex hormones and liver tumors. The human liver expresses estrogen and androgen receptors and experimentally both androgens and estrogens have been implicated in stimulating hepatocyte proliferation and may act as liver tumor inducers or promoters.
I would suggest you actually have an idea of what you are talking about if you are going to try to "correct" me