- Some people said EQ causes some suppression but not full shutdown. What evidence is there for this and is it dose dependent (I figure it must be to some degree but I don’t know the pharmacokinetics of EQ in detail)? Is there a way you could get blood tests at different times on cycle to figure if your dose is suppressing you a little or shutting you down completely?
I’m hearing everything yall are saying and I respect everyones expertise on this board. But this ^ one question wasn’t fully answered. I specifically read on a forum somewhere that EQ causes some suppression but not full shutdown. Something about EQ being mostly anabolic and less androgenic. So I understand full shutdown happening with Tren and Deca but I was left with the impression that EQ was a little different for some reason. I really wondered what the deal was that.
Reed wouldn’t 200mg test per 50mg EQ be way too little EQ? So 1g test would be run with 250mg EQ? I keep reading EQ does nothing at less than 400mg
why would you NOT be suppressed when taking in an additional androgen at doses larger than you naturally produce?
I understand that you would be supressed on EQ. But, some post somewhere said you would not be fully shutdown, as if there was a difference. the exact wording was i believe “some suppression but not full shutdown”, which piqued my curiosity. Whereas running test at even a useless dose like 100mg/wk will of course cause full shutdown immediately.
Again I don’t know pharmacokinetics of EQ in detail. Although I did find on interesting article on ironmagazine which delved into it (mods prolly won’t let me link directly to it) titled Boldenone - realizing its true potential. The author recommends 1.5 - 3 grams per week (!!) with relatively low test dosage. Interesting.
Bottom line though, I understand EQ would be ideal to run with test. But I would prefer to run minimal test. So EQ only cycles came to mind
well, just because someone, somewhere said there is “minimal suppression” doesn’t mean it’s at all accurate (i’m still pissed of at the stupid “half-life” chart that i used to parrot to everybody).
the HPTA is suppressed by an intake of exogenous androgens (which i think you get). taking a bunch of EQ is likely to suppress your HPTA just like an approximate other amount of an androgen. one might assume because it doesn’t affect sex drive like Deca (which is largely a prolactin issue) doesn’t mean that the HPTA isn’t still suppressed.
another concern of a long-acting androgen, is the fact that one remains on it longer, thereby increase the duration of suppression.
the reduced aromatization as compared to testosterone is nice, but that’s not all that hard to manage with an AI.
since EQ binds weakly to the AR, one needs to use more EQ to see an approximate effect to testosterone, which again goes back to suppression, and the effect that a larger amount of androgens can cause.
and the other issue is cost, with EQ being quite a bit more expensive than test, at least where i am…
if you wanna try ti just to try, then go ahead. i just think you’re ignoring a lot of facts about steroids that you prolly already know…