Boxer: I thank you much for allowing me to once again quote myself. I said this in my first post: [quote]“On the contrary, coffee or caffeine ingestion at this time (i.e. before fasted-state cardio or with a protein and fat meal) might actually prove beneficial. This should increase plasma free-fatty acids, and with stable insulin levels, this should bode well since the body will be primed to mobilize those fatty acids right to the muscles’ mitochondria for oxidation.”[/quote]
Vain: That’s an interesting post, but I’m going to stick with my guns and the available literature and side with Joel et al. on this one. In addition to the studies posted by Joel above, here’s one more:
Folch N. P?ronnet F, Massicotte D Duclos M, Lavoie C, Hillaire-Marcel C. Metabolic response to small and large 13C-labelled pasta meals following rest or exercise in man. Bri J. Nutri. (5) 671-680, 2001.
[quote]
These results indicate that: (1) de novo lipogenesis, which plays only a minor role for the disposal of an acute dietary carbohydrate load, is totally suppressed following exercise, even when a very large carbohydrate load is ingested; (2) the reduction in glycogen turnover as well as a preferential conversion of glucose into glycogen are responsible for the increase in glycogen stores following exercise; (3) for a similar energy expenditure, exercise at low workload for a longer period does not favour fat oxidation when the post-exercise period is taken into account.[/quote]
J Am Coll Nutr 1997 Apr;16(2):140-6 Related Articles, Links
Postexercise energy expenditure and substrate oxidation in young women resulting from exercise bouts of different intensity.
Phelain JF, Reinke E, Harris MA, Melby CL.
Department of Food Science and Human Nutrition, Colorado State University, Ft. Collins 80523, USA.
OBJECTIVE: The effects of low and high intensity exercise, of similar energy output, on exercise and post-exercise energy expenditure and substrate oxidation were studied in eight active, eumenorrheic females (aged 22 to 31). METHODS: Continuous indirect calorimetry was performed during cycle ergometry exercise and for 3 hours following each of the following three protocols administered in random order: 1) low intensity exercise (LIE: 500 calories 50% VO2 max), 2) high intensity exercise (HIE: 500 calories 75% VO2 max), and 3) control condition ? of quiet sitting for 1 hour, rather than exercise. Excess postexercise oxygen consumption (EPOC), energy expenditure and total fat and carbohydrate oxidation for the entire exercise/control plus 3-hour recovery period were determined by indirect calorimetry. RESULTS: Mean EPOC for the 3-hour post-exercise period for HIE (9.0 +/- 1.7 L, 41 kcals) was significantly greater than EPOC for low intensity exercise (4.8 +/- 1.6 L, 22 kcals). Oxygen consumption (VO2) following HIE, but not LIE remained elevated at the end of the 3-hour post-exercise period. Total carbohydrate oxidation (exercise plus postexercise period) was significantly higher for HIE (116 +/- 8.6 g) compared to LIE (85.0 +/- 5.2 g). Total fat oxidation was lower for HIE (27.7 +/- 3.3 g) compared to LIE (36.9 +/- 3.0 g), but this difference did not reach statistical significance (p = 0.07). At the end of the 3-hour recovery period, the rate of fat oxidation was higher following HIE compared to LIE. CONCLUSION: These data indicate that the recovery period should also be considered when determining the impact of different exercise intensities on total energy expenditure and fat and carbohydrate utilization in women.
So, that’s just a little evidence to support my stance. But, Vain, I cannot argue with your success:-)
Finally, the Randall Effect has been proven wrong (during exercise). Carbohydrate availability really governs substrate metabolism during exercise. However, this is of little importance because: 1. we’re normally performing high-intensity bouts of exercise, and 2. substrate utilization during exercise isn’t our primary concern.