I think that the comparison between that compound and oral turinabol or Tbol is both fair in that it is similar with the exception of the hydroxyl group instead of a ketone on the 3 carbon and an alpha methylation on the 17 carbon. However, their activities and pharmacological profiles will vary. The tweak on the 3 carbon can be reversed by an enzyme (3B-DH) which is the same enzyme responsible for converting androstenediol to testosterone. The alpha-methylation of the 17 carbon however, will cause the change in shape of the whole ring structure, thereby affecting how the compound interacts with receptors and thus its ability to activate a receptor. This alteration also makes the compound orally bioavailable and in some cases liver toxicity is noted with such compounds.
The comment on absorption was based on the assuming you have the base compound and want to use it orally. If it is the base hormone without the 17aa, then its oral bioavailability is very low.
As for 17-B-hydroxy-2-a-17 dimethyl-5-a-androstan-3one azine, it has two alpha-methylations at the 2 and 17 carbons so it is orally bioavailable, the double bond between the 4 and 5 carbons is reduced (analogous to test to DHT reduction by 5-alphareductase), and i assume that the azine refers to some attempt to make another ring off the A-ring, the likes of winny perhaps? In this case, the oxygen would be removed and replaced by a double bonded nitrogen which was single bonded to another nitrogen which in turn was double bonded to the 2 alpha-methyl carbon. But then there is no need to name the 3one part of the compound.