I’ve heard caber can help a guy go on and on and on. Anybody here know about that? I remember reading something about it but I couldn’t find any info.
I’ve heard this. However, my experience on-cycle is that caber doesn’t seem to change much for me. I typically have more trouble achieving orgasm on cycle than off, and it often takes a lot for me to get to that point even without caber. After adding it in, I typically notice no real difference, nor do I experience much of an increase in libido, which is probably because my libido is pretty well maxed out already anyway.
I would be curious to try it off cycle, and may well do so next month, especially as I’m curious what’s going to happen to my libido and performance after coming off this massive cycle I’ve been on for over 6 months now.
It shouldn’t allow one to go on and on. But it should decrease the refractory period.
[quote]BONEZ217 wrote:
It shouldn’t allow one to go on and on. But it should decrease the refractory period. [/quote]
refractory period = time to get it back up after climax… right?
The libido effects of cabergoline operate on the dopimanergic pathways. Try adding 0.5g selegeline, which causes an increase in dopamine levels. That gets you horny, like when you’re on coke.
These drugs can be hit and miss in the libido department, but that combo usually works in some way.
This one time i was returning to London on a flight from Seattle and i’d been takng a lot of selegiline when i was out there - to help me stay focused/sharp at the conference. The dopamine had built up to a high level and when i got back to London, i was gonna see my girlfriend again after a week. So i took a cabergoline and a cialis with a plan of fucking the living shit out of her. Which i indeed did - i came six times that night in total - and honest to god, i could have gone ALL NIGHT if my girlfriend hadn’t collapsed and fallen asleep out of exhaustion. We both had work the next day so we decided to stop fucking at 3am.
However, while she lay there asleep, i was wired on a dopamine high. Which is very much like an amphetamine high. I think my body was fucked from the 12 time zones i had crossed. I stayed awake for 72 HOURS. That’s three days straight. Whch was crazy cos after i finished fucking my girl i went for a massive hill running workout. And then i wenst straight to work and then i had an evening event to attend at the fucking houses of parliament. And a 2 hour Muay Thai class was squeezed in there.
I thnk getting that levels of sexual performance requires me to really fuck up my dopamine system so i don’t think it’s worth it. So therefore i only dose these drugs at a very low-level. And if i have a lot of testosteron running through me, then the multi-orgasmic side is a lot easier to reach. Serious, thie one time i had a wank and had three full EJACULATORY orgasms in one minute - best internet/weed/porn wank-a-thon ever!
[quote]bushidobadboy wrote:
You sound like you might be into them, if dopamine elevation is your thing - as it is everybodys’ thing, they just don’t realise it, lol.
BBB[/quote]
LOL’ed at this one! = )
[quote]WyldFlower wrote:
The libido effects of cabergoline operate on the dopimanergic pathways. Try adding 0.5g selegeline, which causes an increase in dopamine levels. That gets you horny, like when you’re on coke.
These drugs can be hit and miss in the libido department, but that combo usually works in some way.
This one time i was returning to London on a flight from Seattle and i’d been takng a lot of selegiline when i was out there - to help me stay focused/sharp at the conference. The dopamine had built up to a high level and when i got back to London, i was gonna see my girlfriend again after a week. So i took a cabergoline and a cialis with a plan of fucking the living shit out of her. Which i indeed did - i came six times that night in total - and honest to god, i could have gone ALL NIGHT if my girlfriend hadn’t collapsed and fallen asleep out of exhaustion. We both had work the next day so we decided to stop fucking at 3am.
However, while she lay there asleep, i was wired on a dopamine high. Which is very much like an amphetamine high. I think my body was fucked from the 12 time zones i had crossed. I stayed awake for 72 HOURS. That’s three days straight. Whch was crazy cos after i finished fucking my girl i went for a massive hill running workout. And then i wenst straight to work and then i had an evening event to attend at the fucking houses of parliament. And a 2 hour Muay Thai class was squeezed in there.
I thnk getting that levels of sexual performance requires me to really fuck up my dopamine system so i don’t think it’s worth it. So therefore i only dose these drugs at a very low-level. And if i have a lot of testosteron running through me, then the multi-orgasmic side is a lot easier to reach. Serious, thie one time i had a wank and had three full EJACULATORY orgasms in one minute - best internet/weed/porn wank-a-thon ever![/quote]
Will try at a low dose. I could never get it up on coke or X though. Caused some missed opportunities:^( Haven’t touched it in years.
Thanks guys for your help!
Hagar I cannot speak to the selegiline. But I’ve had some go arounds with Caber both on and off cycle. To address your first point yeah caber does nothing for coming on or off cycle; its not a PCT dryg. Guys use it on cycle primarily to combat prolactin gyno from nor19s. Using on cycle YMMV of course but the longer I’ve been on cycle the less effective I find caber to be for me.
It certainly is better IMO either off cycle in the first few weeks of a cycle. The asterik to that is the dose can and does matter. Average starting point seems to be 1m a week. That is .5mg E3D/twice a week/E4D. There is a correspondence between dose and effectiveness like everything else.
So I’ve heard of guys going up to 4mg a week and having much better results. But caber isnt cheap and so I’ve never tried more than 1mg a week myself. Caber like most things also seems to have a cumulative effect in that 1mg a week will become more effective after you’ve been doing that 5-6 weeks than just your firs or second week.
I can confirm as mentioned above that the Cialis/Caber combo is particularly effective. I also like to take high [5-10g] doses of maca which for me increases cum volume. The three in concert together have give me 4 orgasms in a day as well as the ability to cum stay hard keep pounding away and then cum again a few minutes later. Add some provirion into the mix and I’d almost guarantee results
[quote]saps wrote:
Hagar I cannot speak to the selegiline. But I’ve had some go arounds with Caber both on and off cycle. To address your first point yeah caber does nothing for coming on or off cycle; its not a PCT dryg. Guys use it on cycle primarily to combat prolactin gyno from nor19s. Using on cycle YMMV of course but the longer I’ve been on cycle the less effective I find caber to be for me. It certainly is better IMO either off cycle in the first few weeks of a cycle. The asterik to that is the dose can and does matter. Average starting point seems to be 1m a week. That is .5mg E3D/twice a week/E4D. There is a correspondence between dose and effectiveness like everything else. So I’ve heard of guys going up to 4mg a week and having much better results. But caber isnt cheap and so I’ve never tried more than 1mg a week myself. Caber like most things also seems to have a cumulative effect in that 1mg a week will become more effective after you’ve been doing that 5-6 weeks than just your firs or second week.
I can confirm as mentioned above that the Cialis/Caber combo is particularly effective. I also like to take high [5-10g] doses of maca which for me increases cum volume. The three in concert together have give me 4 orgasms in a day as well as the ability to cum stay hard keep pounding away and then cum again a few minutes later. Add some provirion into the mix and I’d almost guarantee results[/quote]
My proviron’s been doin jack shit in this combination. What dose proviron do you use?
[quote]WyldFlower wrote:
saps wrote:
Hagar I cannot speak to the selegiline. But I’ve had some go arounds with Caber both on and off cycle. To address your first point yeah caber does nothing for coming on or off cycle; its not a PCT dryg. Guys use it on cycle primarily to combat prolactin gyno from nor19s. Using on cycle YMMV of course but the longer I’ve been on cycle the less effective I find caber to be for me. It certainly is better IMO either off cycle in the first few weeks of a cycle. The asterik to that is the dose can and does matter. Average starting point seems to be 1m a week. That is .5mg E3D/twice a week/E4D. There is a correspondence between dose and effectiveness like everything else. So I’ve heard of guys going up to 4mg a week and having much better results. But caber isnt cheap and so I’ve never tried more than 1mg a week myself. Caber like most things also seems to have a cumulative effect in that 1mg a week will become more effective after you’ve been doing that 5-6 weeks than just your firs or second week.
I can confirm as mentioned above that the Cialis/Caber combo is particularly effective. I also like to take high [5-10g] doses of maca which for me increases cum volume. The three in concert together have give me 4 orgasms in a day as well as the ability to cum stay hard keep pounding away and then cum again a few minutes later. Add some provirion into the mix and I’d almost guarantee results
My proviron’s been doin jack shit in this combination. What dose proviron do you use?
[/quote]
50mg
a day? split in two doses? how soon before you feel the pro-libido effects?
[quote]bushidobadboy wrote:
Well selegeline never did this for me, lol.
But your reasoning is fairly sound, though it has to be pointed out that whilst it is the sympathetic NS that controlls ejaculation, it is the parasympathetic NS that controls erection. So, get too wired (on coke, 'phet, ephedrine, caffine or anything that jacks up the sympathetic nervous system) and you’ll struggle to get it up.
BBB[/quote]
so you’re saying if your para-sympathetic nervous system goes into overdrive, it will sabotage your ability to get erections?
Also, i’ve been reading that Aderrall sends your libido off the charts (similar to the experience i had with cabergoline+selegiline). Does that do it through dopamine stimulation too?
OMG BBB, i dunno if i’m going to go down a very dark whole with this shit…
Ok, so i’ve done some more research into this - and correct me if i’m wrong. The multi-orgasmic effects of cabergolie + selegiline happened only twice - and since then i’ve never been able to recreate the intensity. Therefore, something else must have been goig on in my body that complemented that peak.
So you’re saying sympathetic NS mediates orgasm, and para-SNS mediates erection. Over-stimulation of the sympathetic NS causes a “shut-down” of the para-SNS. Furthermore, sympathetic NS is associated with the “fight or flight” response (i.e. arousal of varyin intensity) - whereas the the para-sympathetic controlls the “rest and response” response (this is all wikipedia).
This is why you need to be in a state of calm (para-sympathetic) to achieve erection - which is why psycological anxiety is the biggest cause of Erectile Dysfunction. This is also why some people find it difficult to piss when other people are watching - because urinary function is also controlled by the para-SNS.
Dopamine and norepinephrine are sympathetic NS stimulants and in high doses (when on coke, 'phet, adderall for example) can shut down the para-SNS and therefore prevent erections - even though they, ironically, greatly magnify the sexual excitement response
So, and this is the dark bit. Could stimulating the para-SNS ALONG WITH using sympathetic NS boosters (such as the selegiline + cabergoline combo) induce more reliable multi-orgasms and hard and erect whenever ready response?
I’ve discovered that acetylcholine is the key neurotransmitter for the para-SNS. Just like dopamine/norepinephrine is for the sympathetic NS. It has an effect on the neuro-muscular junctions - therefore is also a potent CNS stimulant - in that it greatly enhances strength. It is also a muscle relaxant and could cause heart complications (chemical weapons and snake/spider poisons contain this to paralyse their pray).
However, could taking a safe dosage of a drug like galantamine/neostigmine give enough of a para-SNS stimulation to COMPLEMENT the symapathetic NS stimulants?
These drugs are used to traet Alzheimers patients and are widely available (galantamine is sold as a supplement) and both increase acetylcholine concentrations. This is important for preserving the neuro-plasticity of the brain in helping you learn new things and adapt.
So i would add neostigimine to my stack and thereby get both sympathetic and para-sympathetic NS stimulation.
Is that crazy?
Woah, check this out!
Method of reducing psychological impotence
This invention relates neostigmine bromide or neostigmine methyl sulfate as a pharmaceutical agent to reduce psychogenic impotence in physiologically normal men.
Men who are physiologically normal by all available tests but who fail to achieve erections in most of their attempts have been treated almost exclusively by psychological means with relatively small success. Such erectile impotence has long been observed to have a significant association with worry and anxiety.
It is the primary object of this invention to provide a pharmaceutical treatment for psychological or psychogenic impotence.
Neostigmine bromide or neostigmine methyl sulfate which is available commercially as Prostigmin bromide or Prostigmin methyl sulfate from Roche Pharmaceutical Co. stimulates the parasympathetic nervous system. It inhibits the destruction of acetylcholine by inhibiting or inactivating acetylcholinerase, the enzyme which normally destroys naturally formed and released acetylcholine; thereby, neostigmine bromide allows additional cholinergic stimulation. Thus, neostigmine bromide causes acetylcholine to accumulate and to exert a longer stimulatory action at sensitive sites throughout the nervous system.
For this reason, the principal use of neostigmine bromide is in the treatment of Myasthenia Gravis whose basic symptom is muscular weakness which results from a reduction in the amount of acetylcholine available at the nerve-muscle junction. It is the acetylcholine which causes the muscle to respond by contraction. In Myasthenia Gravis, neostigmine bromide seems also to act directly on the muscle membrane, thereby enhancing muscular strength. The aerage daily dose of neostigmine bromide in the treatment of Myasthenia Gravis is 150 mg (ten 15 mg tablets). Some patients receive as much as 400 mg neostigmine bromide daily.
The actions of the sympathetic and parasympathetic nervous systems are opposed. The sympathetic nervous system seems to prepare the body for nervous or vigorous muscular activity and has therefore been called the system of “flight or fight”; whereas the parasympathetic nervous system acts more discretely on individual organs or regions of the body and has therefore been called the “housekeeper” of the body, regulating ordinary functions of living (digestion, urine flow, etc.). Accordingly, the integrating activity of the autonomic nervous system (sympathetic and parasympathetic) is of vital importance for the well being of the organism.
Applicant believes that neostigmine bromide is a balancing agent for these two opposed systems because of its cholinergic stimulating effect on the parasympathetic nervous system. While neostigmine bromide has been used for paralytic ileus and atony of the urinary bladder, glaucoma and some other eye conditions, and atropine intoxication as reported in “The Pharmacological Basis of Therapeutics” by Goodman and Gilman (1975 Ed.) Macmillan & Company, applicant has discovered that in relatively small doses and by oral mucosal absorption, neostigmine bromide is very effective in reducing psychological or psychogenic impotence in men.
Applicant is aware that neostigmine bromide has been used intrathecally, i.e. by injection into the part of the back associated with outflow of nerves to the genitalia from the spinal cord, in cord-injured patients to produce ejaculation generally with erection, Guttman and Walsh: “Prostigmin Assessment Test of Fertility in Spinal Man”, Paraplegia 9:39-51, 1971 and Griffith, Tomko and Timms, “Sexual Function in Spinal Cord-Injured Patients: A Review” Arch. Phys. Med. Rehabil. Vol. 54, pp. 539-543, Dec. 1973.
However, it is applicant who has discovered the effectiveness of neostigmine bromide on physiologically normal but psychologically impotent men especially when administered by oral mucosal absorption, i.e. sub-lingually.
A number of men were treated with neostigmine bromide in accordance with the instant invention and the results obtained are shown in the following Table 1. All men treated had an essentially normal physical examination and were not taking any other drugs or medicines, aside from coffee or tea. Blood flow to their genitals was clinically adequate and all had been previously diagnosed as “psychologically” impotent.
Thus, it has been discovered that administering neostigmine bromide or neostigmine methyl sulfate in relatively small doses of 15 mg or 30 mg without one hour prior to sexual attempt, especially when administered by swilling or oral mucosal absorption (sub-lingually) has a significant affect on diminishing psychological impotence. In the sub-lingual administration, the neostigmine bromide takes effect in 8-10 minutes. Since it increases the parasympathetic tone by cholinergic stimulation and since older people may produce too little choline, administration of neostigmine bromide may enhance the sexuality in older people.
or even combining bupropion + selegeline + cabergolie + neostigmine + cialis + SERM + Maca + proviron for a stack of sexual excess.
could be a recipe for Priapism
Also check this for the pro-erectile effects of galanthamine - also an acetylcholine stimulator of the para-SNS
http://www.patentstorm.us/patents/5177070/description.html
"Physiologic erectile impotence differs from psychogenic erectile impotence in significant ways and one can readily separate the two types. From early childhood through at least the eighties, erections occur during normal sleep and is known as nocturnal penile tumescence or NPT. This happens during rapid eye movement (REM) sleep and the total NPT time per night averages about 100 min. These erections continue to occur in patients having the psychogenic form of erectile impotence while they do not occur in men with physiological
causes for their impotence. Therefore, the simple observation that erectile function is present during sleep indicates that a psychogenic cause rather than a physiologic cause is at work.
As stated above, galanthamine, a cholinergic drug believed potentially to be useful for Senile Demetia of the Alzheimer’s Type (SDAT), has been reported to alleviate the psychogenic form of erectile impotence.
Physostigmine has been used to obtain an ejaculation from paraplegic men (Andrologia 20 (4):311-3, July-August 1988). Neostigmine has been used intrathecally to obtain a sample from a paraplegic male for artificial insemination purposes (Paraplegia 24 (1):32-7, 1986 Feburary).
The United States Pharmacopoeia, 9th Edition lists papaverine and phentolamine as useful in impotence. Papaverine is an opium alkaloid and it relaxes smooth muscle in the ureter and blood vessels. Phentolamine is an adrenergic inhibitor, while physostigmine and neostigmine are cholinergic agents."
So these “cholinergic” agents seem to combat the “psychological” causes of ED, in that they stimulate the relaxation response…