I need some of the best minds to comment on this if that’s possible.
Is it possible for an adult over 40, to have gotten the pathology osteolysis, due to AAS in any way shape or form? Being as they are “anabolic”, vs catabolic. Now I know normal bone growth is over at the weening age of lads, but if one were to be so predispositioned in some weird way to aquiring this bone softening/hypertrophy disease, would, or could, AAS such as Test, in some strange manner, perhaps “catalyze” such a pathology?
I was told by an expert,(my surgeon), that this common weightlifters disease is caused by very heavy benching and shoulder pressing. The forces focus right on the distal clavicle and the most popular theory is that microfractures occur in the end of the bone, and somehow initiate the softening/enlarging process of said clavicle. Won’t get into it more than that, but…
I’m curious as hell, as I look to see the surgeon again tomorrow, since I have it back after about a year and a few months or so post surgery for the initial diagnosis and bone buzzing which set me free for this length of time anyway from the disease.
If you have any insight, please drop it on me.
Btw, I’m aware that indeed they would contribute to the “cause” indirectly by virtue of strength increase, and thus increased forces acting upon said clav. I’m wondering if they physiologically induce or aggravate the pathology somehow.