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Bone Remodeling From Use of AAS?

I need some of the best minds to comment on this if that’s possible.

Is it possible for an adult over 40, to have gotten the pathology osteolysis, due to AAS in any way shape or form? Being as they are “anabolic”, vs catabolic. Now I know normal bone growth is over at the weening age of lads, but if one were to be so predispositioned in some weird way to aquiring this bone softening/hypertrophy disease, would, or could, AAS such as Test, in some strange manner, perhaps “catalyze” such a pathology?

I was told by an expert,(my surgeon), that this common weightlifters disease is caused by very heavy benching and shoulder pressing. The forces focus right on the distal clavicle and the most popular theory is that microfractures occur in the end of the bone, and somehow initiate the softening/enlarging process of said clavicle. Won’t get into it more than that, but…

I’m curious as hell, as I look to see the surgeon again tomorrow, since I have it back after about a year and a few months or so post surgery for the initial diagnosis and bone buzzing which set me free for this length of time anyway from the disease.

If you have any insight, please drop it on me.

Btw, I’m aware that indeed they would contribute to the “cause” indirectly by virtue of strength increase, and thus increased forces acting upon said clav. I’m wondering if they physiologically induce or aggravate the pathology somehow.

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[quote]bushidobadboy wrote:
Kind of edit:

Since DCO would appear to be a product of overactive osteoclasts and fibroblasts, and underactive osteoblasts, you might try discovering more about the paracrine signalling hormones that stimulate/inhibit these things. Perhaps some sort of localised injection - maybe alflutops? - would help. Although the AC joint is not synovial in construction, I wonder if some of the same destructive enzymes are present in its lysis.


Well, shit me a wagon full of fucking cheer mate!!
Ha, haaa, just got back from the surgeon, and Santa saved my ass it looks like! No further remodeling has occurred at all! The initial gap of approx 1 cm, is still intact at just that distance! Fantastic news!
I’m literally just blown away with happiness.

Pisses me right off that the general practioner told me that it WAS back, and remodeling HAD occurred, when no such thing had at all!? This is the same guy who misdiagnosed it over a year and a half ago. Had said cortisone shot, and two weeks I’d be benching heavy back then…Yeah right!

I had only told him I thought it was back as I had felt that same pressure on that clav/acro area, and that I had really gone for it etc in the gym. Simply to get the xrays authorized…
I mean, why on God’s earth would I have him “advise” me at this point, when my surgeon who is tops in the area already has me as a patient? He had me really freaked…

Bottom line is, thank you big A for answering my question. You made some good points about looking into paracrines, and the like as you stated. You’re always thinking ahead of the curve…:slight_smile:

So color this thread answered and disposed of then.

Somebody say trenny run? Mwahaha…

SO fucking glad it’s not back. Time to get back and make up for 2 mos. lost time in the pit on upper body.

Thanks again BBB, I knew I could count on you droppin some brainflow on the subject as always!


To answer my own question then, now that I’ve seen the xrays, consulted with an expert surgeon, etc.

Apparently AAS will not necessarily initiate or directly cause this pathology. Proof being as how I myself have been on a few things here and there during the past year, they in fact don’t seem to even indirectly cause it either. My gap sustained it’s original distance during all these heavy workouts.

From what I’ve gathered, the pressure I felt, in that same area must be something else, possibly just a strain on what was left of my ligament between the two bones.

Just thought I might as well address the original question I posed with this new info.

The space or lack of it, between the clavicle and the acromium, is mainly what decides if it is to commence into full blown dco, or not, along with the amount of heavy benching or military style pressing one does.

I had a very tight space, then that coupled initially with the DC style of training, put the thing in motion. So I hope that explains what was going on here for anyone who might have been interested…

Later dudes, and many thanks for your input again Al.