For the men here I urge you to think with logic not experience alone. Be open to evidence. Use logic not fear to make decisions when doing trt.
For those who love to tell men to block estrogen anytime a symptom arises. Or if the estrogen lab range is high. Please read this article. Provide similar evidence disputing these facts. It won’t happen. @hrdlvn you especially love to do this and I hope I can convince you otherwise. Until now you have not once had a gentlemanly debate on this subject. You always disappear when the questions get tough. Yet you run around telling folks “this forum is not checked for good advice and Be careful who you listen to”. Yet you yourself have not once provided an ounce or shred of evidence supporting your guidance of taking ai and being scared of estrogen.
For the new men. If someone suggests anything related to Trt do not take anyone’s word for it. Find literature and evidence that backs up their comments. If they cannot
Backup their suggestions I suggest you put any suggestions of theirs out your mind.
In regards to estrogen in men the conclusion of this study below. Read page 3 and we find that estrogen is producing many of the effects we look for when starting trt. Men with low estrogen and high T are having issues. Men wirh low t and high estrogen are having issues. How do we find the balance ? Let the body do its magic folks. I
The benefits of TrT are simple: theee hormones in one are produced. Without this synergy we are losing a third or more of our benefits.
Anyone here who thinks e2 should be blocked please provide a study that shows this.
I WILL NOTE THAT estrogen symptoms are non existent when free t , DHT and estrogen have a good balance. Most lower trt doses when symptoms arise. Instead of trying more they continually lower their dose because everyome thinks estrogen. Next time try more and a higher frequency like daily.
Remember that the body aromitizes for a reason. It needs this hormone. Why screw with your bodily functions?
Also another study I will post in the next comment about estrogen and how it’s produced and why we were wrong all along thinking a e2 sensitive test is accurate.
Recent evidence has demonstrated that many biological actions historically attributed to testosterone are instead, at least in part, mediated by its aromatization product E2. The data are strongest for effects on bone, fat mass, insulin resistance and VMS. The relevance of these data is that clinically efficacious treatment of male hypogonadism is best achieved with testosterone, which provides ‘three hormones in one’ – testosterone, DHT, E2. Conversely, this evidence raises caution regarding the use of selective androgen receptor modulators, non- aromatizable androgens and AIs for male hypogonadism, and emphasizes the need for better understanding of the tissue-specific effects of SERMs, which are also used off label by some practitioners for this purpose. They also suggest that E2, either as sole ADT or as add-back to conventional GnRH analog-based ADT, may be a promising treatment to mitigate some of the adverse effects of ADT given to men with prostate cancer. Most current studies in men are relatively small, short term, and the design of experimental studies does not always recapitulate physiology. More research is needed