When e2 elevated it causes a rise in TBG which makes the thyroid less functionable. As thyroid lower this triggers elevated Norephinperine which puts you in a state of heighten response (figh or flight response) There are also may be adrenal imbalances here as well which needs to be ruled out through salivary cortisol profile. Once you understand how the systems integrate it is not hard coming up with the reason why some thing may be occuring.
You can be hyper or normal in the blood and low in the tissue from low cortisol or low ferritin levels pooling the thyroid in the blood. Please check adrenals through 4 point cortisol saliva test and ferritin levels.
KS man I agree. I am in the process of getting the E2 taken care of as we speak. Just started my adex a couple days ago. I'm taking 2mg/ day for 3 days to get it knocked down. Would you reccomend longer on the 2mg?
Another part of the problem is my doc gave me the shitty generic thyroid meds which I tried to explain to him make me feel shitty. I basically have to take double the dose to feel normal. So with high E2 and crappy thyroid meds I've ended up w/ a double effect of what hardasnails described in the first response. I am slowly coming out of the fog, but still get spells anxiety spells although less severe adn much shorter.
yep, had highish TSH 2+ and low ft3. took 120mg Armour for awhile, but ran out one day (pretty common with me and my adhd) and I didn't notice a single thing different from being off armour, so I didn't get it refilled. did some research and learned about RT3. Ran some tests and found out that I was over the max.
I first worked on supporting my low cortisol levels by taking 5 mg hydrocortisone and slowly worked up to the 15-20mg range. then I started on T3 only medication and built up to 30mg daily. now I am keeping my body temps and waiting a couple of months to retest and see where my blood levels are.
Purechance, Have you checked your shbg? When ever treating thyroid please address adrenals first through 24 hour cortisol saliva. Using t3 directly works for a small percentage of people. Idenifying why your rt3 is elevated and working to reduce it at the root cause would be the appropriate approach.