^that study refers to a reduction in the number of androgen receptors. not really binding, but it does affect their number...
that is interesting to see, although that study was referencing the effect of E2 on the AR in breast cancer cells.
anyway, i found this, which claims E cannot bind to the AR:
"Hormones, though, only bind to certain, compatible receptor types. Thyroid hormones and each steroid hormone group - the estrogens, androgens, progestins, glucocorticoids (stress hormones), and mineralocorticoids (water and ion-regulating hormones) - have a matching hormone receptor type. For instance, estrogen hormones, like estrone, bind to estrogen receptors (ER); androgen hormones, such as testosterone, bind to androgen receptors; and so on.
But, steroid hormone receptors for each hormone group can occur in several versions that differ in form, function, and location. So, steroid and thyroid hormones are not restricted to interact with only one receptor, in one tissue, to produce one kind of action. The hormones can easily bind and activate several versions of their matching receptor type. An example is the estrogen 17-beta-estradiol. It binds to both the ER alpha and ER beta receptors but not to androgen, progestin, or thyroid receptors."
however, i'm gonna keep doing some digging, as i think you have any interesting point here, as far as reducing androgen receptors...